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About the database ScholarIQanswers from OpenAlex & ORCID
How has Gerd Walz's publication output changed over time?
ScholarIQpublication output · 1990–2012
Output grew0% over the shown period — from 1 works in 1990 to 1 in 2012.
1
1
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1
2
1
2
1
1
199019972003200520062008201020112012
What are the most-cited papers on Gerd Walz?
ScholarIQmost cited works
Recognition by Elam-1 of the Sialyl-Le <sup>x</sup> Determinant on Myeloid and Tumor Cells
Gerd Walz, Alejandro Aruffo, Waldemar Kolanus, M P Bevilacqua, Brian Seed
Science. 1990969 Citations
The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease
Jonathan M. Shillingford, Noel Murcia, Claire H. Larson, Seng Hui Low, Ryan C. Hedgepeth, Nicole Brown, Chris A. Flask, Andrew C. Novick, David A. Goldfarb, Albrecht Kramer-Zucker, Gerd Walz, Klaus Piontek, Gregory G. Germino, Thomas Weimbs
Proceedings of the National Academy of Sciences. 2006800 CitationsOPEN ACCESS
Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways
Matias Simons, Joachim Gloy, Athina Ganner, Axel Bullerkotte, Mikhail Bashkurov, Corinna Krönig, Bernhard Schermer, Thomas Benzing, Olga A. Cabello, Andreas Jenny, Marek Mlodzik, Bozena Polok, Wolfgang Driever, Tomoko Obara, Gerd Walz
S137905309. 2005763 Citations
Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice
Björn Hartleben, Markus Gödel, Catherine Meyer‐Schwesinger, Shuya Liu, Theresa A. Ulrich, Sven Köbler, Thorsten Wiech, Florian Grahammer, Sebastian J. Arnold, Maja T. Lindenmeyer, Clemens D. Cohen, Hermann Pavenstädt, Dontscho Kerjaschki, Noboru Mizushima, Andréy S. Shaw, Gerd Walz, Tobias B. Huber
S114430552. 2010715 CitationsOPEN ACCESS
Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination
Edgar A. Otto, Bernhard Schermer, Tomoko Obara, John F. O’Toole, Karl S. Hiller, Adelheid M. Mueller, Rainer Ruf, Julia Hoefele, Frank Beekmann, Daniel Landau, John W. Foreman, Judith A. Goodship, Tom Strachan, Andreas Kispert, Matthias T. F. Wolf, M. F. Gagnadoux, Hubert Nivet, Corinne Antignac, Gerd Walz, Iain A. Drummond, Thomas Benzing, Friedhelm Hildebrandt
S137905309. 2003651 CitationsOPEN ACCESS
Related on ScholarIQ
University of Freiburg
Institution
Recognition by Elam-1 of the Sialyl-Le <sup>x</sup> Determinant on Myeloid and Tumor Cells
Paper
The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease
Paper
Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways
Paper
Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice
Paper
Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination
Paper