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About the database ScholarIQanswers from OpenAlex & ORCID
How has Hong Zhu's publication output changed over time?
ScholarIQpublication output · 1994–2024
Output grew0% over the shown period — from 1 works in 1994 to 1 in 2024.
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199419982000200320072012201620182024
What are the most-cited papers on Hong Zhu?
ScholarIQmost cited works
Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis
Honglin Li, Hong Zhu, Chi-jie Xu, Junying Yuan
S110447773. 19984,445 CitationsOPEN ACCESS
Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β
Toshiyuki Nakagawa, Hong Zhu, Nobuhiro Morishima, En Li, Jin Xu, Bruce A. Yankner, Junying Yuan
Nature. 20003,268 Citations
Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death
Lin Wang, Masayuki Miura, Louise Bergeron, Hong Zhu, Junying Yuan
S110447773. 1994840 Citations
Murine Caspase-11, an ICE-Interacting Protease, Is Essential for the Activation of ICE
Suyue Wang, Masayuki Miura, Yong‐Keun Jung, Hong Zhu, En Li, Junying Yuan
S110447773. 1998701 CitationsOPEN ACCESS
RIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS
Yasushi Ito, Dimitry Ofengeim, Ayaz Najafov, Sudeshna Das, Shahram Saberi, Ying Li, Junichi Hitomi, Hong Zhu, Hongbo Chen, Lior Mayo, Jiefei Geng, Palak Amin, Judy Park DeWitt, Adnan K. Mookhtiar, Marcus A. Florez, Amanda Tomie Ouchida, Jian-Bing Fan, Manolis Pasparakis, Michelle A. Kelliher, John Ravits, Junying Yuan
Science. 2016617 CitationsOPEN ACCESS
Related on ScholarIQ
Jiangsu University
Institution
Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis
Paper
Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β
Paper
Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death
Paper
Murine Caspase-11, an ICE-Interacting Protease, Is Essential for the Activation of ICE
Paper
RIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS
Paper